Table S1. Sequences of the primers used in this study.docx
Table S2. Phosphopeptides with significantly altered abundance in seedlings overexpressing individual AEL genes (AEL1-OE, AEL2-OE, AEL3-OE, AEL4-OE) and lacking function of one or more AELs.xlsx
Table S3. Distribution of phosphorylated sites among serine, threonine, and tyrosine in AELs-overexpression or ael mutants lines compared to Col-0.xlsx
Table S4. AEL-upregulated phosphopeptides with higher abundance in AEL-OE lines relative to Col-0, or lower abundance in ael mutants compared to Col-0, respectively.xlsx
Table S5. AEL-downregulated phosphopeptides with lower abundance in AEL-OE lines relative to Col-0, or higher abundance in ael mutants compared to Col-0, respectively.xlsx
Table S6. Phosphoproteins corresponding to AEL-upregulated phosphopeptides were identified as putative substrates.xlsx
Table S7. Putative substrate proteins of AELs in Arabidopsis predicted with the motifs identified in this study.xlsx
Table S8. Functional analysis of total putative substrates of AELs in Arabidopsis with Gene ontology (GO) annotation.xlsx
Table S9. Putative substrate proteins of CK1s in rice predicted with the motifs identified in this study.xlsx
Table S10. Functional analysis of total putative substrates of CK1s in rice with Gene ontology (GO) annotation.xlsx
Table S11. Putative substrate proteins of CK1s in mice predicted with the motifs identified in this study.xlsx
Table S12. Functional analysis of total putative substrates of CK1s in mice with Gene ontology (GO) annotation.xlsx
Table S13. Putative substrate proteins of CK1s in humans predicted with the motifs identified in this study.xlsx
Table S14. Functional analysis of total putative substrates of CK1s in humans with Gene ontology (GO) annotation.xlsx
Table S15. Conserved CK1 functions in both Arabidopsis and humans.xlsx
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